Live Quiz Arena
🎁 1 Free Round Daily
⚡ Enter ArenaQuestion
← Human Body & HealthA patient on long-term antibiotics develops neurological symptoms including ataxia and confusion. Which mechanism explains why these symptoms arise following antibiotic use in this context?
A)Increased gut iron absorption
B)Impaired thiamine synthesis✓
C)Enhanced vitamin K production
D)Reduced folate bioavailability
💡 Explanation
Antibiotics disrupt the gut microbiome, which reduces the production of certain vitamins. This can lead to impaired thiamine synthesis by bacteria, which is essential for neuronal function; therefore, neurological symptoms arise, rather than increased iron absorption or altered production of vitamin K or folate.
🏆 Up to £1,000 monthly prize pool
Ready for the live challenge? Join the next global round now.
*Terms apply. Skill-based competition.
Related Questions
Browse Human Body & Health →- Why does thyroid hormone (TH) production often remain stable despite variations in thyrotropin-releasing hormone (TRH) from the hypothalamus?
- Following a mRNA vaccine booster, which mechanism explains the enhanced antibody response compared to the primary series, demonstrating long-term immunological memory?
- Which consequence arises when tetanus toxin inhibits synaptic vesicles' fusion?
- Why does a high-fiber meal promote satiety via GLP-1 secretion in the gut?
- Why does sensorineural hearing loss often exhibit frequency-dependent sensitivity?
- Why does hypertension damage blood vessel walls more rapidly than in normotensive individuals?
