An individual experiences recurrent luteal phase defects despite normal ovulation. Which compensatory mechanism masks the defect's impact on early pregnancy?

💡 Explanation

Human chorionic gonadotropin (hCG) supplementation mimics luteinizing hormone (LH) and supports progesterone production, rescuing the corpus luteum. This occurs because hCG stimulates the LH receptor on luteal cells, therefore compensating for inadequate progesterone secretion rather than increasing FSH or endometrial prostaglandins.

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