If a patient's renal tubules chronically reabsorb excessive oxalate, which consequence is most likely, assuming normal kidney function otherwise?

💡 Explanation

Excessive oxalate reabsorption increases its concentration in the urine, promoting supersaturation and thus calcium oxalate crystallization because increased oxalate drives the reaction. Therefore, kidney stone formation increases, rather than phosphate stone formation or changes in filtration rate or uric acid precipitation.

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